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Access through your institution Buy or subscribe The human intestinal pathogen _Shigella flexneri_ negotiates its way through the crowded interior of host cells by destroying the surrounding
microtubule network, according to a recent _Science_ paper. Some bacterial pathogens invade the cytoplasm of infected host cells to establish a protective niche and allow the infection to
spread to neighbouring cells. Following invasion, _S. flexneri_, _Listeria monocytogenes_ , _Burkholderia pseudomallei_ , _Mycobacterium marinum_ and the spotted-fever Rickettsiae all
manipulate the host-cell cytoskeleton, recruiting actin and inducing its polymerization at one end of the bacterial cell to generate the propulsion necessary for motility. This is a preview
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ADDITIONAL ACCESS OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support REFERENCES ORIGINAL RESEARCH PAPER * Yoshida, S. et al.
Microtubule-severing activity of _Shigella_ is pivotal for intercellular spreading. _Science_ 314, 985–989 (2006) Article CAS Google Scholar FURTHER READING * Stevens, J. M., Galyov, E.
E. & Stevens, M. P. Actin-dependent movement of bacterial pathogens. _Nature Rev. Microbiol._ 4, 91–101 (2006) Article CAS Google Scholar Download references Authors * Sheilagh Molloy
View author publications You can also search for this author inPubMed Google Scholar RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Molloy, S. Clearing
a path. _Nat Rev Microbiol_ 5, 7 (2007). https://doi.org/10.1038/nrmicro1584 Download citation * Issue Date: 01 January 2007 * DOI: https://doi.org/10.1038/nrmicro1584 SHARE THIS ARTICLE
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