- Select a language for the TTS:
- UK English Female
- UK English Male
- US English Female
- US English Male
- Australian Female
- Australian Male
- Language selected: (auto detect) - EN
Play all audios:
ABSTRACT In the simple, 302-cell nervous system of the soil nematode _C. elegans_, genetic selection for toxin-resistance was used to isolate mutants with specific defects in synaptic
neurotransmission, making it possible to characterize already-known molecules and to identify new components of the machinery required for the release of synaptic vesicles. Access through
your institution Buy or subscribe This is a preview of subscription content, access via your institution ACCESS OPTIONS Access through your institution Subscribe to this journal Receive 12
print issues and online access $259.00 per year only $21.58 per issue Learn more Buy this article * Purchase on SpringerLink * Instant access to full article PDF Buy now Prices may be
subject to local taxes which are calculated during checkout ADDITIONAL ACCESS OPTIONS: * Log in * Learn about institutional subscriptions * Read our FAQs * Contact customer support AUTHOR
INFORMATION AUTHORS AND AFFILIATIONS * Program in Molecular and Cell Biology, Oklahoma Medical Research Foundation, 825 NE 13th St, Oklahoma City, 73104, OK, USA J B Rand Authors * J B Rand
View author publications You can also search for this author inPubMed Google Scholar RIGHTS AND PERMISSIONS Reprints and permissions ABOUT THIS ARTICLE CITE THIS ARTICLE Rand, J. What makes
the worm squirm?. _Mol Psychiatry_ 2, 293–295 (1997). https://doi.org/10.1038/sj.mp.4000244 Download citation * Received: 24 September 1996 * Accepted: 14 November 1996 * Issue Date: 01 July
1997 * DOI: https://doi.org/10.1038/sj.mp.4000244 SHARE THIS ARTICLE Anyone you share the following link with will be able to read this content: Get shareable link Sorry, a shareable link
is not currently available for this article. Copy to clipboard Provided by the Springer Nature SharedIt content-sharing initiative