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THE cell wall preparation from Saccharomyces cerevisiae, designated as zymosan, has been reported to possess a variety of biological activities which include the ability, in as yet an
undefined manner, to induce an increase in the functional activity of reticulo-endothelial cells1–4. The hyperphagocytic state was associated with pronounced hypertrophy of organs containing
predominantly reticulo-endothelial cells4,5. Based on the incorporation of tritium-labelled thymidine into hepatic DNA following a single injection of zymosan, the hyperplasia was observed
to be the result of a specific reticulo-endothelial or littoral cell proliferation2.
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